In Vivo Modelling of ATP1A3 G316S-Induced Ataxia in C. elegans Using CRISPR/Cas9-Mediated Homologous Recombination Reveals Dominant Loss of Function Defects.

Sorkac A, Alcantara IC, Hart AC
PLoS One. 2016 Dec 9;11(12):e0167963. doi: 10.1371/journal.pone.0167963. eCollection 2016. PubMed Article

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