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Annexins A1 and A2 are recruited to larger lysosomal injuries independently of ESCRTs to promote repair.

Yim WW, Yamamoto H, Mizushima N
FEBS Lett. 2022 Mar 11. doi: 10.1002/1873-3468.14329. (Link opens in a new window) PubMed (Link opens in a new window) Article

Plasmids from Article

ID Plasmid Purpose
184888pMRX-IZ-ANXA1-mRuby3Stable expression of ANXA1-mRuby3 (Annexin A1-mRuby3) in mammalian cells
184889pMRX-IZ-ANXA2-mRuby3Stable expression of ANXA2-mRuby3 (Annexin A2-mRuby3) in mammalian cells
184890pMRX-IZ-ANXA4-mRuby3Stable expression of ANXA4-mRuby3 (Annexin A4-mRuby3) in mammalian cells
184891pMRX-IZ-ANXA5-mRuby3Stable expression of ANXA5-mRuby3 (Annexin A5-mRuby3) in mammalian cells
184892pMRX-IZ-ANXA6-mRuby3Stable expression of ANXA6-mRuby3 (Annexin A6-mRuby3) in mammalian cells
184893pMRX-IZ-ANXA7-mRuby3Stable expression of ANXA7-mRuby3 (Annexin A7-mRuby3) in mammalian cells
184894pMRX-IZ-ANXA11-mRuby3Stable expression of ANXA11-mRuby3 (Annexin A11-mRuby3) in mammalian cells
184895pMRX-IZ-HaloTag7-CHMP4AStable expression of HaloTag7-CHMP4A in mammalian cells
184896pMRX-IZ-HaloTag7-Galectin3Stable expression of HaloTag7-Galectin3 in mammalian cells
184897pMRX-IB-TMEM192-mGFPStable expression of TMEM192-mGFP in mammalian cells

Antibodies from Article