GFP-Bcl2-Maob Citations (4)

Originally described in: Transient expression of wild-type or mitochondrially targeted Bcl-2 induces apoptosis, whereas transient expression of endoplasmic reticulum-targeted Bcl-2 is protective against Bax-induced cell death.
Wang NS, Unkila MT, Reineks EZ, Distelhorst CW J Biol Chem. 2001 Nov 23. 276(47):44117-28. PubMed Journal

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Articles Citing GFP-Bcl2-Maob

Articles
Threonine 56 phosphorylation of Bcl-2 is required for LRRK2 G2019S-induced mitochondrial depolarization and autophagy. Su YC, Guo X, Qi X. Biochim Biophys Acta. 2015 Jan;1852(1):12-21. doi: 10.1016/j.bbadis.2014.11.009. Epub 2014 Nov 15. PubMed
Gene delivery ability of polyethylenimine and polyethylene glycol dual-functionalized nanographene oxide in 11 different cell lines. Wu L, Xie J, Li T, Mai Z, Wang L, Wang X, Chen T. R Soc Open Sci. 2017 Oct 25;4(10):170822. doi: 10.1098/rsos.170822. eCollection 2017 Oct. PubMed
HIV-1 Nef counteracts autophagy restriction by enhancing the association between BECN1 and its inhibitor BCL2 in a PRKN-dependent manner. Castro-Gonzalez S, Shi Y, Colomer-Lluch M, Song Y, Mowery K, Almodovar S, Bansal A, Kirchhoff F, Sparrer K, Liang C, Serra-Moreno R. Autophagy. 2021 Feb;17(2):553-577. doi: 10.1080/15548627.2020.1725401. Epub 2020 Feb 25. PubMed
TOM20-mediated transfer of Bcl2 from ER to MAM and mitochondria upon induction of apoptosis. Lalier L, Mignard V, Joalland MP, Lanoe D, Cartron PF, Manon S, Vallette FM. Cell Death Dis. 2021 Feb 15;12(2):182. doi: 10.1038/s41419-021-03471-8. PubMed

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