pCCL cPPT PGK EGFP WPRE LTR H1 shSOD1mismatch Citations (3)
Originally described in: Lentiviral-mediated silencing of SOD1 through RNA interference retards disease onset and progression in a mouse model of ALS.Raoul C, Abbas-Terki T, Bensadoun JC, Guillot S, Haase G, Szulc J, Henderson CE, Aebischer P Nat Med. 2005 Apr . 11(4):423-8. PubMed Journal
Articles Citing pCCL cPPT PGK EGFP WPRE LTR H1 shSOD1mismatch
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Aberrant association of misfolded SOD1 with Na(+)/K(+)ATPase-alpha3 impairs its activity and contributes to motor neuron vulnerability in ALS. Ruegsegger C, Maharjan N, Goswami A, Filezac de L'Etang A, Weis J, Troost D, Heller M, Gut H, Saxena S. Acta Neuropathol. 2016 Mar;131(3):427-51. doi: 10.1007/s00401-015-1510-4. Epub 2015 Nov 30. PubMed |
Notch Signaling Activation in Cervical Cancer Cells Induces Cell Growth Arrest with the Involvement of the Nuclear Receptor NR4A2. Sun L, Liu M, Sun GC, Yang X, Qian Q, Feng S, Mackey LV, Coy DH. J Cancer. 2016 Jun 30;7(11):1388-95. doi: 10.7150/jca.15274. eCollection 2016. PubMed |
Activated Notch signaling augments cell growth in hepatocellular carcinoma via up-regulating the nuclear receptor NR4A2. Zhu B, Sun L, Luo W, Li M, Coy DH, Yu L, Yu W. Oncotarget. 2017 Apr 4;8(14):23289-23302. doi: 10.18632/oncotarget.15576. PubMed |
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