pLJC6-3XHA-TMEM192 Citations (4)

Originally described in: mTORC1 Activator SLC38A9 Is Required to Efflux Essential Amino Acids from Lysosomes and Use Protein as a Nutrient.
Wyant GA, Abu-Remaileh M, Wolfson RL, Chen WW, Freinkman E, Danai LV, Vander Heiden MG, Sabatini DM Cell. 2017 Oct 19;171(3):642-654.e12. doi: 10.1016/j.cell.2017.09.046. PubMed Journal

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Articles Citing pLJC6-3XHA-TMEM192

Articles
An Epstein-Barr virus protein interaction map reveals NLRP3 inflammasome evasion via MAVS UFMylation. Yiu SPT, Zerbe C, Vanderwall D, Huttlin EL, Weekes MP, Gewurz BE. Mol Cell. 2023 Jul 6;83(13):2367-2386.e15. doi: 10.1016/j.molcel.2023.05.018. Epub 2023 Jun 12. PubMed
Loss of the batten disease protein CLN3 leads to mis-trafficking of M6PR and defective autophagic-lysosomal reformation. Calcagni' A, Staiano L, Zampelli N, Minopoli N, Herz NJ, Di Tullio G, Huynh T, Monfregola J, Esposito A, Cirillo C, Bajic A, Zahabiyon M, Curnock R, Polishchuk E, Parkitny L, Medina DL, Pastore N, Cullen PJ, Parenti G, De Matteis MA, Grumati P, Ballabio A. Nat Commun. 2023 Jul 3;14(1):3911. doi: 10.1038/s41467-023-39643-7. PubMed
Glycerophosphodiesters inhibit lysosomal phospholipid catabolism in Batten disease. Nyame K, Hims A, Aburous A, Laqtom NN, Dong W, Medoh UN, Heiby JC, Xiong J, Ori A, Abu-Remaileh M. Mol Cell. 2024 Apr 4;84(7):1354-1364.e9. doi: 10.1016/j.molcel.2024.02.006. Epub 2024 Mar 5. PubMed
Glucose starvation causes ferroptosis-mediated lysosomal dysfunction. Miki K, Yagi M, Kang D, Kunisaki Y, Yoshimoto K, Uchiumi T. iScience. 2024 Apr 12;27(5):109735. doi: 10.1016/j.isci.2024.109735. eCollection 2024 May 17. PubMed

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